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This process, coupled with the exhaustion of myeloid and lymphoid cells, synergistically induces a significant upregulation in the production of pro-inflammatory cytokines, including interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-)
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Integrin 51FAK/Src signaling is activated by alteration in the ECM composition, which in turn enhances the motility, while lymphocyte infiltration is restricted by the dense collagen network, adding another layer of immunosuppression (113)
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